What Regulatory Bodies and Experts Say About GLP-1 Plus Phentermine The American Academy of Clinical Endocrinologists (AACE) and The Obesity Society have not issued formal position statements endorsing GLP-1 combined with phentermine, reflecting clinical equipoise and safety uncertainty
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It has multiple actions including: potentiation of glucose-mediated insulin secretion mechanisms identified: increased -cell proliferation, resulting in an increase -cell mass (Fusco et al, 2017) stimulation of insulin biosynthesis at the translational level, helping to maintain -cell insulin stores and secretory capacity (Baggio & Drucker, 2007) because the GLP-1 effect is glucose-dependent (there is more insulin release when glucose levels are elevated, but less effect when glucose levels are normal), GLP-1 agonists have a lower risk for producing hypoglycemia compared to sulfonylureas (that chronically stimulate insulin release, independent of glucose concentration) suppression of postprandial glucagon release Evidence indicates that stimulation of pancreatic cells by GLP-1 increases their glucose sensitivity, resulting in less glucagon release at any glucose level (Baggio & Drucker, 2007)
Some individuals achieve significant melanogenesis at 0.5mg daily, while others require 0.751.0mg
